for patients

Mechanism of action

The targets – phosphatidyl serine

The phospholipid composition of the stressed cancer cell membranes is deregulated: phosphatidyl serine (PS) from the inside moves into the outer layer of the cell membrane. The extremely high affinity of PS and H1 for each other leads to the formation of PS-H1 aggregates and - in a second step - of superaggregates which form channels in the membrane. These channels enable the uncontrolled mass exchange between the cell and the outside, leading to the rapid death of the cell (apoptosis followed by necrosis).

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